A Putative Mechanism of Age-Related Synaptic Dysfunction Based on the Impact of IGF-1 Receptor Signaling on Synaptic CaMKIIα Phosphorylation-Reference-Cited by-同舟云学术

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A Putative Mechanism of Age-Related Synaptic Dysfunction Based on the Impact of IGF-1 Receptor Signaling on Synaptic CaMKIIα Phosphorylation

Published:2018-05-14 Issue: Volume:12 Page:
ISSN:1662-5129
Container-title:Frontiers in Neuroanatomy
language:
Short-container-title:Front. Neuroanat.

Author:

Ogundele Olalekan M.,Pardo Joaquin,Francis Joseph,Goya Rodolfo G.,Lee Charles C.

Funder

International Brain Research Organization

National Institutes of Health

Louisiana Board of Regents

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience,Neuroscience (miscellaneous),Anatomy

Reference92 articles.

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2. Synaptic and intrinsic mechanisms shape synchronous oscillations in hippocampal neurons in culture;Bacci;Eur. J. Neurosci.,1999

3. Insulin-like growth factor 1 (IGF-1) and aging: controversies and new insights;Bartke;Biogerontology,2003

4. Compensatory enlargement of synaptic size in aging and senile dementia;Bertoni-Freddari;Boll. Soc. Ital. Biol. Sper.,1993

5. Enlargement of synaptic size as a compensative reaction in aging and dementia;Bertoni-Freddari;Pathol. Res. Pract.,1992

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