The Key Role of c-Fos for Immune Regulation and Bacterial Dissemination in Brucella Infected Macrophage-Reference-Cited by-同舟云学术

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The Key Role of c-Fos for Immune Regulation and Bacterial Dissemination in Brucella Infected Macrophage

Published:2018-08-21 Issue: Volume:8 Page:
ISSN:2235-2988
Container-title:Frontiers in Cellular and Infection Microbiology
language:
Short-container-title:Front. Cell. Infect. Microbiol.

Author:

Hop Huynh T.,Arayan Lauren T.,Huy Tran X. N.,Reyes Alisha W. B.,Vu Son H.,Min WonGi,Lee Hu J.,Rhee Man H.,Chang Hong H.,Kim Suk

Publisher

Frontiers Media SA

Subject

Infectious Diseases,Microbiology (medical),Immunology,Microbiology

Reference50 articles.

1. Toll-like receptor 4 expression is required to control chronic Mycobacterium tuberculosis infection in mice;Abel;J. Immunol,2002

2. TLR signaling is required for Salmonella typhimurium virulence;Arpaia;Cell,2011

3. Helicobacter pylori induces ERK-dependent formation of a phosphor-c-Fos c-Jun activator protein 1 complex that cause apoptosis in macrophages;Asim;J. Biol. Chem,2010

4. A cardinal role for cathepsin d in co-ordinating the host-mediated apoptosis of macrophages and killing of pneumococci;Bewley;PLoS. Pathog.,2011

5. Brucella evades macrophage killing via VirB-dependent sustained interactions with the endoplasmic reticulum;Celli;J. Exp. Med.,2003

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