APOBEC3G-Mediated G-to-A Hypermutation of the HIV-1 Genome: The Missing Link in Antiviral Molecular Mechanisms
Author:
Funder
Japan Society for the Promotion of Science
Publisher
Frontiers Media SA
Subject
Microbiology (medical),Microbiology
Reference91 articles.
1. Retroviral restriction factor APOBEC3G delays the initiation of DNA synthesis by HIV-1 reverse transcriptase.;Adolph;PLoS ONE,2013
2. Incomplete APOBEC3G/F neutralization by HIV-1 Vif mutants facilitates the genetic evolution from CCR5 to CXCR4 usage.;Alteri;Antimicrob. Agents Chemother.,2015
3. APOBEC3G restricts early HIV-1 replication in the cytoplasm of target cells.;Anderson;Virology,2008
4. Promiscuous RNA binding ensures effective encapsidation of APOBEC3 proteins by HIV-1.;Apolonia;PLoS Pathog.,2015
5. APOBEC3G-induced hypermutation of human immunodeficiency virus type-1 is typically a discrete “all or nothing” phenomenon.;Armitage;PLoS Genet,2012
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