PARK2 Mutation Causes Metabolic Disturbances and Impaired Survival of Human iPSC-Derived Neurons-Reference-Cited by-同舟云学术

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PARK2 Mutation Causes Metabolic Disturbances and Impaired Survival of Human iPSC-Derived Neurons

Published:2019-07-05 Issue: Volume:13 Page:
ISSN:1662-5102
Container-title:Frontiers in Cellular Neuroscience
language:
Short-container-title:Front. Cell. Neurosci.

Author:

Bogetofte Helle,Jensen Pia,Ryding Matias,Schmidt Sissel I.,Okarmus Justyna,Ritter Louise,Worm Christina S.,Hohnholt Michaela C.,Azevedo Carla,Roybon Laurent,Bak Lasse K.,Waagepetersen Helle,Ryan Brent J.,Wade-Martins Richard,Larsen Martin R.,Meyer Morten

Funder

Innovationsfonden

Lundbeckfonden

Parkinsonforeningen

Jascha Fonden

Fonden til Lægevidenskabens Fremme

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience

Reference51 articles.

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2. A generalised increase in protein carbonyls in the brain in Parkinson’s but not incidental Lewy body disease.;Alam;J. Neurochem.,1997

3. New insights on the mitochondrial proteome plasticity in Parkinson’s disease.;Aroso;Proteomics Clin. Appl.,2016

4. High levels of mitochondrial DNA deletions in substantia nigra neurons in aging and Parkinson disease.;Bender;Nat. Genet.,2006

5. Functional interplay between Parkin and Drp1 in mitochondrial fission and clearance.;Buhlman;Biochim. Biophys. Acta,2014

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4. Induced-pluripotent stem cells and neuroproteomics as tools for studying neurodegeneration;Biochemical Society Transactions;2024-01-30

5. USP30 inhibition induces mitophagy and reduces oxidative stress in parkin-deficient human neurons;Cell Death & Disease;2024-01-15

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