Intracellular Trafficking Mechanisms of Synaptic Dysfunction in Alzheimer’s Disease-Reference-Cited by-同舟云学术

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Intracellular Trafficking Mechanisms of Synaptic Dysfunction in Alzheimer’s Disease

Published:2020-04-17 Issue: Volume:14 Page:
ISSN:1662-5102
Container-title:Frontiers in Cellular Neuroscience
language:
Short-container-title:Front. Cell. Neurosci.

Author:

Perdigão Catarina,Barata Mariana A.,Araújo Margarida N.,Mirfakhar Farzaneh S.,Castanheira Jorge,Guimas Almeida Cláudia

Funder

Fundação para a Ciência e a Tecnologia

Alzheimer’s Association

H2020 Spreading Excellence and Widening Participation

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience

Reference236 articles.

1. Amyloid-β as a positive endogenous regulator of release probability at hippocampal synapses;Abramov;Nat. Neurosci.,2009

2. ABCA7 loss-of-function variants, expression, and neurologic disease risk;Allen;Neurol. Genet.,2017

3. β-amyloid accumulation impairs multivesicular body sorting by inhibiting the ubiquitin-proteasome system;Almeida;J. Neurosci.,2006

4. β-amyloid accumulation in APP mutant neurons reduces PSD-95 and GluR1 in synapses;Almeida;Neurobiol. Dis.,2005

5. 2019 Alzheimer’s disease facts and figures;Alzheimers Dement.,2019

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