Case 150: Van Buchem Disease (Hyperostosis Corticalis Generalisata)-Reference-Cited by-同舟云学术

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Case 150: Van Buchem Disease (Hyperostosis Corticalis Generalisata)

Published:2009-10 Issue:1 Volume:253 Page:272-276
ISSN:0033-8419
Container-title:Radiology
language:en
Short-container-title:Radiology

Author:

Wengenroth Martina,Vasvari Gergely,Federspil Philipp A.,Mair Johanna,Schneider Peter,Stippich Christoph

Publisher

Radiological Society of North America (RSNA)

Subject

Radiology Nuclear Medicine and imaging

Link

http://pubs.rsna.org/doi/pdf/10.1148/radiol.2531080011

Reference19 articles.

1. A generalized skeletal hyperostosis in two siblings caused by a novel mutation in the gene

2. Differential diagnosis and treatment of autosomal dominant osteosclerosisof the mandible

3. Albers-Schonberg disease (autosomal dominant osteopetrosis, type II) results from mutations in the ClCN7 chloride channel gene

4. Heterogeneity of autosomal dominant osteopetrosis.

5. Sclerosing bone disorders

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1. Inhibiting WNT secretion reduces high bone mass caused by Sost loss-of-function or gain-of-function mutations in Lrp5;Bone Research;2023-08-24

2. Inhibiting Wnt Secretion Reduces High Bone Mass Caused by Sost Deficiency or Point Mutations in Lrp5;2023-01-10

3. Posterior Vault Distraction for Multi-Suture Craniosynostosis in a Patient with Craniometaphyseal Dysplasia: A Case Report;Craniomaxillofacial Research & Innovation;2022-01

4. Van Buchem Disease: First Case Report from the Indian Subcontinent with an Early Presentation;Journal of Child Science;2021-01

5. Sclerostin Antibody–Induced Changes in Bone Mass Are Site Specific in Developing Crania;Journal of Bone and Mineral Research;2019-11-07

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