Three-Way Translocation t(12;15;17) (p13;q24;q21) Found in Acute Promyelocytic Leukemia with Basophilic Differentiation

Author:

Frazzetto Sara12,Gullo Lara12ORCID,Sapuppo Gabriele12,Fazio Manlio12ORCID,Lo Faro Cristina12,Giunta Giuliana12,Caravotta Ignazio12,Mauro Elisa1,Parisi Marina Silvia1,Triolo Anna Maria1,Parrinello Nunziatina Laura1,Consoli Maria Letizia1,També Loredana1,Cambria Daniela1,Marino Sara1,Scuderi Grazia1,Di Raimondo Francesco12

Affiliation:

1. Division of Hematology, Azienda Policlinico-San Marco, 95123 Catania, Italy

2. Postgraduate School of Hematology, University of Catania, 95123 Catania, Italy

Abstract

Acute promyelocytic leukemia is a rare form of acute myeloid leukemia in which immature promyelocytes abnormally proliferate in the bone marrow. In most cases, the disease is characterised by the translocation t(15;17) (q24;q21), which causes the formation of PML::RARA, an oncogenic fusion protein responsible for blocking myeloid differentiation and survival advantage. Here, we present a case of acute promyelocytic leukemia with two unusual features: basophilic differentiation and a three-way translocation involving chromosomes 12, 15 and 17. In the few cases reported, basophilic differentiation was associated with a poor prognosis. In contrast, our patient responded promptly to the standard treatment with all-trans-retinoic acid (ATRA) and arsenic trioxide (ATO) and obtained complete remission. To our knowledge, this is the first report of basophilic acute promyelocytic leukemia with the three-way translocation t(12;17;15) (p13; q24;q21).

Publisher

MDPI AG

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