COVID-19 Infection May Drive EC-like Myofibroblasts towards Myofibroblasts to Contribute to Pulmonary Fibrosis
Author:
Affiliation:
1. Division of Cardiology, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095, USA
2. Department of Pediatrics, Stanford University, Stanford, CA 94305, USA
3. The Molecular Biology Institute at UCLA, Los Angeles, CA 90095, USA
Abstract
Funder
National Institute of Neurological Disorders and Stroke
National Heart Lung and Blood Institute
Publisher
MDPI AG
Subject
Inorganic Chemistry,Organic Chemistry,Physical and Theoretical Chemistry,Computer Science Applications,Spectroscopy,Molecular Biology,General Medicine,Catalysis
Link
https://www.mdpi.com/1422-0067/24/14/11500/pdf
Reference24 articles.
1. Clinical features of patients infected with 2019 novel coronavirus in Wuhan, China;Huang;Lancet,2020
2. European Respiratory Society statement on long COVID follow-up;Antoniou;Eur. Respir J.,2022
3. Pulmonary fibrosis secondary to COVID-19: A call to arms?;Spagnolo;Lancet Respir. Med.,2020
4. Global incidence and mortality of idiopathic pulmonary fibrosis: A systematic review;Hutchinson;Eur. Respir. J.,2015
5. Idiopathic Pulmonary Fibrosis;Lederer;N. Engl. J. Med.,2018
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Three-dimensional cultured human umbilical cord mesenchymal stem cells attenuate pulmonary fibrosis by improving the balance of mitochondrial fusion and fission;Stem Cells Translational Medicine;2024-07-30
2. Reply: Transitioning endothelial cells contribute to pulmonary fibrosis;European Respiratory Journal;2023-09
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