Correlation between Genomic Variants and Worldwide COVID-19 Epidemiology

Author:

Costa Ana Caroline Alves da1ORCID,Albarello Gellen Laura Patrícia1ORCID,Fernandes Marianne Rodrigues12ORCID,Coelho Rita de Cássia Calderaro1,Monte Natasha1,Moraes Francisco Cezar Aquino de1ORCID,Calderaro Maria Clara Leite1,Freitas Lilian Marques de1,Matos Juliana Aires1,Fernandes Thamara Fernanda da Silva1,Aguiar Kaio Evandro Cardoso1ORCID,Vinagre Lui Wallacy Morikawa Souza12ORCID,dos Santos Sidney Emanuel Batista13,dos Santos Ney Pereira Carneiro1

Affiliation:

1. Oncology Research Center, Federal University of Pará, Belém 66073-005, PA, Brazil

2. Ophir Loyola Hospital, Pará State Departament of Health, Belém 66063-240, PA, Brazil

3. Laboratory of Human and Medical Genetics, Institute of Biological Science, Federal University of Pará, Belém 66077-830, PA, Brazil

Abstract

COVID-19 is a systemic disease caused by the etiologic agent SARS-CoV-2, first reported in Hubei Province in Wuhan, China, in late 2019. The SARS-CoV-2 virus has evolved over time with distinct transmissibility subvariants from ancestral lineages. The clinical manifestations of the disease vary according to their severity and can range from asymptomatic to severe. Due to the rapid evolution to a pandemic, epidemiological studies have become essential to understand and effectively combat COVID-19, as the incidence and mortality of this disease vary between territories and populations. This study correlated epidemiological data on the incidence and mortality of COVID-19 with frequencies of important SNPs in GWAS studies associated with the susceptibility and mortality of this disease in different populations. Our results indicated significant correlations for 11 genetic variants (rs117169628, rs2547438, rs2271616, rs12610495, rs12046291, rs35705950, rs2176724, rs10774671, rs1073165, rs4804803 and rs7528026). Of these 11 variants, 7 (rs12046291, rs117169628, rs1073165, rs2547438, rs2271616, rs12610495 and rs35705950) were positively correlated with the incidence rate, these variants were more frequent in EUR populations, suggesting that this population is more susceptible to COVID-19. The rs2176724 variant was inversely related to incidence rates; therefore, the higher the frequency of the allele is, the lower the incidence rate. This variant was more frequent in the AFR population, which suggests a protective factor against SARS-CoV-2 infection in this population. The variants rs10774671, rs4804803, and rs7528026 showed a significant relationship with mortality rates. SNPs rs10774671 and rs4804803 were inversely related to mortality rates and are more frequently present in the AFR population. The rs7528026 variant, which is more frequent in the AMR population, was positively related to mortality rates. The study has the potential to identify and correlate the genetic profile with epidemiological data, identify populations that are more susceptible to severe forms of COVID-19, and relate them to incidence and mortality.

Funder

CNPq

FAPESPA

CAPES

UFPA

Publisher

MDPI AG

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