Distinct Fiber Type Signature in Mouse Muscles Expressing a Mutant Lamin A Responsible for Congenital Muscular Dystrophy in a Patient
Author:
Funder
Association Française contre les Myopathies
Publisher
MDPI AG
Subject
General Medicine
Link
http://www.mdpi.com/2073-4409/6/2/10/pdf
Reference31 articles.
1. Nuclear lamins: major factors in the structural organization and function of the nucleus and chromatin
2. Lamins: Nuclear Intermediate Filament Proteins with Fundamental Functions in Nuclear Mechanics and Genome Regulation
3. “Laminopathies”: A wide spectrum of human diseases
4. Mouse models of laminopathies
5. Viral-mediated expression of desmin mutants to create mouse models of myofibrillar myopathy
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Proteomic characterization of human LMNA-related congenital muscular dystrophy muscle cells;Neuromuscular Disorders;2024-05
2. Differential histological features and myogenic protein levels in distinct muscles of d-sarcoglycan null muscular dystrophy mouse model;Journal of Molecular Histology;2023-06-26
3. Histological Methods to Assess Skeletal Muscle Degeneration and Regeneration in Duchenne Muscular Dystrophy;International Journal of Molecular Sciences;2022-12-16
4. CYB5R3 overexpression preserves skeletal muscle mitochondria and autophagic signaling in aged transgenic mice;GeroScience;2022-05-09
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3