Differences between Mice and Humans in Regulation and the Molecular Network of Collagen, Type III, Alpha-1 at the Gene Expression Level: Obstacles that Translational Research Must Overcome-Reference-Cited by-同舟云学术

Differences between Mice and Humans in Regulation and the Molecular Network of Collagen, Type III, Alpha-1 at the Gene Expression Level: Obstacles that Translational Research Must Overcome

Published:2015-07-03 Issue:12 Volume:16 Page:15031-15056
ISSN:1422-0067
Container-title:International Journal of Molecular Sciences
language:en
Short-container-title:IJMS

Author:

Wang Lishi,Liu Hongchao,Jiao Yan,Wang Erjian,Clark Stephen,Postlethwaite Arnold,Gu Weikuan,Chen Hong

Publisher

MDPI AG

Subject

Inorganic Chemistry,Organic Chemistry,Physical and Theoretical Chemistry,Computer Science Applications,Spectroscopy,Molecular Biology,General Medicine,Catalysis

Link

http://www.mdpi.com/1422-0067/16/7/15031/pdf

Reference30 articles.

1. A base substitution at a splice site in the COL3A1 gene causes exon skipping and generates abnormal type III procollagen in a patient with Ehlers-Danlos syndrome type IV;Cole;J. Biol. Chem.,1990

2. Haploinsufficiency for One COL3A1 Allele of Type III Procollagen Results in a Phenotype Similar to the Vascular Form of Ehlers-Danlos Syndrome, Ehlers-Danlos Syndrome Type IV

3. Type III collagen is crucial for collagen I fibrillogenesis and for normal cardiovascular development

4. The Haploinsufficient Col3a1 Mouse as a Model for Vascular Ehlers-Danlos Syndrome

5. Transcriptional activation of α1(III) procollagen gene in Tsk2/+ dermal fibroblasts

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