The Heat-Stable Enterotoxin Receptor, Guanylyl Cyclase C, as a Pharmacological Target in Colorectal Cancer Immunotherapy: A Bench-to-Bedside Current Report-Reference-Cited by-同舟云学术

The Heat-Stable Enterotoxin Receptor, Guanylyl Cyclase C, as a Pharmacological Target in Colorectal Cancer Immunotherapy: A Bench-to-Bedside Current Report

Published:2017-09-15 Issue:9 Volume:9 Page:282
ISSN:2072-6651
Container-title:Toxins
language:en
Short-container-title:Toxins

Author:

Baybutt Trevor,Aka Allison,Snook Adam

Publisher

MDPI AG

Subject

Health, Toxicology and Mutagenesis,Toxicology

Link

http://www.mdpi.com/2072-6651/9/9/282/pdf

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1. Nivolumab in patients with metastatic DNA mismatch repair-deficient or microsatellite instability-high colorectal cancer (CheckMate 142): an open-label, multicentre, phase 2 study

2. Microsatellite Instability in Colorectal Cancer

3. A review of serious adverse effects under treatment with checkpoint inhibitors

4. Oncogenic cancer/testis antigens: prime candidates for immunotherapy

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1. Enterotoxin‐related genes PPFIA4 and SCN3B promote colorectal cancer development and progression;Journal of Biochemical and Molecular Toxicology;2024-05-20

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4. Preclinical Chimeric Antibody Chimeric Antigen Receptor T Cell Progress in Digestive System Cancers;Cancer Biotherapy and Radiopharmaceuticals;2021-01-22

5. TCR Retrogenic Mice as a Model To Map Self-Tolerance Mechanisms to the Cancer Mucosa Antigen GUCY2C;The Journal of Immunology;2019-01-14