The Biased G-Protein-Coupled Receptor Agonism Bridges the Gap between the Insulin Receptor and the Metabolic Syndrome-Reference-Cited by-同舟云学术

The Biased G-Protein-Coupled Receptor Agonism Bridges the Gap between the Insulin Receptor and the Metabolic Syndrome

Published:2018-02-17 Issue:2 Volume:19 Page:575
ISSN:1422-0067
Container-title:International Journal of Molecular Sciences
language:en
Short-container-title:IJMS

Author:

Liauchonak Iryna,Dawoud Fady,Riat Yatin,Qorri Bessi,Sambi Manpreet,Jain Justin,Kalaydina Regina-Veronicka,Mendonza Nicole,Bajwa Komal,Szewczuk Myron

Publisher

MDPI AG

Subject

Inorganic Chemistry,Organic Chemistry,Physical and Theoretical Chemistry,Computer Science Applications,Spectroscopy,Molecular Biology,General Medicine,Catalysis

Link

http://www.mdpi.com/1422-0067/19/2/575/pdf

Reference112 articles.

1. Insulin Induces Heterologous Desensitization of G Protein-Coupled Receptor and Insulin-Like Growth Factor I Signaling by Downregulating β-Arrestin-1

2. A Comprehensive Review on Metabolic Syndrome

3. Biochemical and cellular properties of insulin receptor signalling

4. The insulin receptor: a prototype for dimeric, allosteric membrane receptors?

5. Insulin and Insulin-like Growth Factor I Receptors Utilize Different G Protein Signaling Components

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