Natural Anti-Estrogen Receptor Alpha Antibodies Able to Induce Estrogenic Responses in Breast Cancer Cells: Hypotheses Concerning Their Mechanisms of Action and Emergence-Reference-Cited by-同舟云学术

Natural Anti-Estrogen Receptor Alpha Antibodies Able to Induce Estrogenic Responses in Breast Cancer Cells: Hypotheses Concerning Their Mechanisms of Action and Emergence

Published:2018-01-30 Issue:2 Volume:19 Page:411
ISSN:1422-0067
Container-title:International Journal of Molecular Sciences
language:en
Short-container-title:IJMS

Author:

Leclercq Guy^ORCID

Publisher

MDPI AG

Subject

Inorganic Chemistry,Organic Chemistry,Physical and Theoretical Chemistry,Computer Science Applications,Spectroscopy,Molecular Biology,General Medicine,Catalysis

Link

http://www.mdpi.com/1422-0067/19/2/411/pdf

Reference62 articles.

1. Autoantibodies to estrogen receptors and their involvement in autoimmune diseases and cancer

2. Autoantibodies specific to estrogen receptor alpha act as estrogen agonists and their levels correlate with breast cancer cell proliferation

3. A Subpopulation of Immunoglobulin G in Man Selectively Interacts with the Hormone-Binding Sites of Estrogen Receptors*

4. Estrogen-Like Activity of a Subpopulation of Natural Antiestrogen Receptor Autoantibodies in Man*

5. Natural Antiestrogen Receptor Autoantibodies in Man with Estrogenic Activity in Mammary Carcinoma Cell Culture: Study of their Mechanism of Action; Evidence for Involvement of Estrogen-Like Epitopes1

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1. Antibodies and anti-antibodies specific to estradiol and progesterone and tumor proliferation in breast cancer patients;Siberian journal of oncology;2024-07-03

2. Pathological Maintenance and Evolution of Breast Cancer: The Convergence of Irreversible Biological Actions of ER Alpha;Endocrines;2020-12-24

3. Inhibition of Wnt3a/FOXM1/β-Catenin Axis and Activation of GSK3β and Caspases are Critically Involved in Apoptotic Effect of Moracin D in Breast Cancers;International Journal of Molecular Sciences;2018-09-10

4. Molecular Pathways of Estrogen Receptor Action;International Journal of Molecular Sciences;2018-08-31