Targeting of Both the c-Met and EGFR Pathways Results in Additive Inhibition of Lung Tumorigenesis in Transgenic Mice-Reference-Cited by-同舟云学术

Targeting of Both the c-Met and EGFR Pathways Results in Additive Inhibition of Lung Tumorigenesis in Transgenic Mice

Published:2010-12-22 Issue:4 Volume:2 Page:2153-2170
ISSN:2072-6694
Container-title:Cancers
language:en
Short-container-title:Cancers

Author:

Stabile Laura P.,Rothstein Mary E.,Keohavong Phouthone,Lenzner Diana,Land Stephanie R.,Gaither-Davis Autumn L.,Kim K. Jin,Kaminski Naftali,Siegfried Jill M.

Publisher

MDPI AG

Subject

Cancer Research,Oncology

Link

http://www.mdpi.com/2072-6694/2/4/2153/pdf

Reference30 articles.

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2. Epidermal growth factor receptor overexpression correlates with a poor prognosis in completely resected non-small-cell lung cancer

3. Activating Mutations in the Epidermal Growth Factor Receptor Underlying Responsiveness of Non–Small-Cell Lung Cancer to Gefitinib

4. EGFR Mutations in Lung Cancer: Correlation with Clinical Response to Gefitinib Therapy

5. Acquired Resistance of Lung Adenocarcinomas to Gefitinib or Erlotinib Is Associated with a Second Mutation in the EGFR Kinase Domain

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