Role of Protein Quality Control Failure in Alcoholic Hepatitis Pathogenesis
Author:
Publisher
MDPI AG
Subject
Molecular Biology,Biochemistry
Link
http://www.mdpi.com/2218-273X/7/1/11/pdf
Reference32 articles.
1. Ufmylation and FATylation pathways are downregulated in human alcoholic and nonalcoholic steatohepatitis, and mice fed DDC, where Mallory–Denk bodies (MDBs) form
2. The role of cytokines in UbD promoter regulation and Mallory-Denk body-like aggresomes
3. SAMe prevents the induction of the immunoproteasome and preserves the 26S proteasome in the DDC-induced MDB mouse model
4. FAT10 knock out mice livers fail to develop Mallory–Denk bodies in the DDC mouse model
5. Mallory–Denk Body (MDB) formation modulates ufmylation expression epigenetically in alcoholic hepatitis (AH) and non-alcoholic steatohepatitis (NASH)
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1. SQSTM1/p62 and Hepatic Mallory-Denk Body Formation in Alcohol-Associated Liver Disease;The American Journal of Pathology;2023-10
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3. Quercetin in Tartary Buckwheat Induces Autophagy against Protein Aggregations;Antioxidants;2021-07-29
4. Altered regulation of LncRNA analysis of human alcoholic hepatitis with Mallory-Denk Bodies (MDBs) is revealed by RNA sequencing;Experimental and Molecular Pathology;2020-12
5. Cardiac and vascular changes in the patho- and morphogenesis of alcohol use disorder;Cardiovascular Therapy and Prevention;2020-11-14
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