Acetylated Deoxynivalenol Generates Differences of Gene Expression that Discriminate Trichothecene Toxicity
Author:
Publisher
MDPI AG
Subject
Health, Toxicology and Mutagenesis,Toxicology
Link
http://www.mdpi.com/2072-6651/8/2/42/pdf
Reference36 articles.
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2. Induction of apoptosis and cytokine production in the Jurkat human T cells by deoxynivalenol: role of mitogen-activated protein kinases and comparison to other 8-ketotrichothecenes
3. Comprehensive Gene Expression Analysis of Type B Trichothecenes
4. A genome-wide screen in Saccharomyces cerevisiae reveals a critical role for the mitochondria in the toxicity of a trichothecene mycotoxin
5. Structure−Activity Relationships of Trichothecene Toxins in an Arabidopsis thaliana Leaf Assay
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1. Identification of an Acinetobacter pittii acyltransferase involved in transformation of deoxynivalenol to 3-acetyl-deoxynivalenol by transcriptomic analysis;Ecotoxicology and Environmental Safety;2023-09
2. Targeted Analysis of Sphingolipids in Turkeys Fed Fusariotoxins: First Evidence of Key Changes That Could Help Explain Their Relative Resistance to Fumonisin Toxicity;International Journal of Molecular Sciences;2022-02-24
3. Combination of Antimicrobial Starters for Feed Fermentation: Influence on Piglet Feces Microbiota and Health and Growth Performance, Including Mycotoxin Biotransformation in vivo;Frontiers in Veterinary Science;2020-10-16
4. Nucleic acid amplification free biosensors for pathogen detection;Biosensors and Bioelectronics;2020-04
5. Deoxynivalenol induces toxicity and apoptosis in piglet hippocampal nerve cells via the MAPK signaling pathway;Toxicon;2018-12
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