Foreword Special Issue Genomic Instability in Tumor Evolution and Therapy Response
Author:
Affiliation:
1. Department of Genetics, Physical Anthropology and Animal Physiology, University of the Basque Country, UPV/EHU, 48080 Bilbao, Spain
2. Ikerbasque, Basque Foundation for Science, 48009 Bilbao, Spain
Abstract
Publisher
MDPI AG
Subject
Cancer Research,Oncology
Link
https://www.mdpi.com/2072-6694/15/12/3080/pdf
Reference12 articles.
1. Replication Stress and Cancer;Gaillard;Nat. Rev. Cancer,2015
2. ATM, ATR, and DNA-PK: The Trinity at the Heart of the DNA Damage Response;Blackford;Mol. Cell,2017
3. Pladevall-Morera, D., Castejón-Griñán, M., Aguilera, P., Gaardahl, K., Ingham, A., Brosnan-Cashman, J.A., Meeker, A.K., and Lopez-Contreras, A.J. (2022). ATRX-Deficient High-Grade Glioma Cells Exhibit Increased Sensitivity to RTK and PDGFR Inhibitors. Cancers, 14.
4. The Impact of Replication Stress on Replication Dynamics and DNA Damage in Vertebrate Cells;Koundrioukoff;Nat. Rev. Genet.,2017
5. Hamidi, M., Eriz, A., Mitxelena, J., Fernandez-Ares, L., Aurrekoetxea, I., Aspichueta, P., Iglesias-Ara, A., and Zubiaga, A.M. (2022). Targeting E2F Sensitizes Prostate Cancer Cells to Drug-Induced Replication Stress by Promoting Unscheduled CDK1 Activity. Cancers, 14.
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