Alterations and Co-Occurrence of C-MYC, N-MYC, and L-MYC Expression are Related to Clinical Outcomes in Various Cancers-Reference-Cited by-同舟云学术

Alterations and Co-Occurrence of C-MYC, N-MYC, and L-MYC Expression are Related to Clinical Outcomes in Various Cancers

Published:2023-04-30 Issue:2 Volume:16 Page:215-233
ISSN:2005-5447
Container-title:International Journal of Stem Cells
language:en
Short-container-title:Int J Stem Cells

Author:

Lee Moonjung¹²,Seok Jaekwon¹,Saha Subbroto Kumar¹,Cho Sungha¹,Jeong Yeojin¹,Gil Minchan¹,Kim Aram³,Shin Ha Youn⁴,Bae Hojae¹,Do Jeong Tae¹,Kim Young Bong⁴,Cho Ssang-Goo¹

Affiliation:

1. Department of Stem Cell and Regenerative Biotechnology & Institute of Advanced Regenerative Science, Konkuk University, Seoul, Korea

2. Department of Advanced Translational Medicine, Konkuk University, Seoul, Korea

3. Department of Urology, Konkuk University Medical Center, Konkuk University School of Medicine, Seoul, Korea

4. Department of Biomedical Science & Engineering, KU Convergence Science and Technology Institute, Konkuk University, Seoul, Korea

Funder

Konkuk University

Publisher

Korean Society for Stem Cell Research

Subject

Cell Biology,Developmental Biology

Link

http://pdf.medrang.co.kr/Ijsc2/2023/016/ijsc-16-2-215.pdf

Reference50 articles.

1. Myc family oncoproteins function through a common pathway to transform normal cells in culture: cross-interference by Max and trans-acting dominant mutants.

2. Mnt–Max to Myc–Max complex switching regulates cell cycle entry

3. Induction of Pluripotent Stem Cells from Adult Human Fibroblasts by Defined Factors

4. A null c-myc mutation causes lethality before 10.5 days of gestation in homozygotes and reduced fertility in heterozygous female mice.

5. Loss of N-myc function results in embryonic lethality and failure of the epithelial component of the embryo to develop.

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