Polymorphism of Delta-Aminolevulinic Acid Dehydratase and Its Effect on Blood Lead Levels in Thai Workers
Author:
Affiliation:
1. a The Faculty of Medicine, Ramathibodi Hospital, Mahidol University, Bangkok, Thailand
2. b Mahidol University's Faculty of Graduate Studies
Publisher
Informa UK Limited
Subject
Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health,General Environmental Science,Toxicology
Link
https://www.tandfonline.com/doi/pdf/10.3200/AEOH.61.2.67-72
Reference34 articles.
1. Potluri VR, Astrin KH, Wetmur JG, Bishop DF, Desnick RJ. Human delta-aminolevulinate dehydratase: chromosomal localization to 9q34 by in situ hybridization. Hum Genet. 1987;76:236-239.
2. Kaya AH, Plewinska M, Wong DM, Desnick RJ, Wetmur JG. Human delta-aminolevulinate dehydratase (ALAD) gene: structure and alternative splicing of the erythroid and housekeeping mRNAs. Genomics. 1994;19:242-248.
3. Wetmur JG, Kaya AH, Plewinska M, Desnick RJ. Molecular characterization of the human delta-aminolevulinate dehydratase 2 (ALAD2) allele: implications for molecular screening of individuals for genetic susceptibility to lead poisoning. Am J Hum Genet. 1991;49:757-763.
4. Wetmur JG, Lehnert G, Desnick RJ. The delta-aminolevulinate dehydratase polymorphism: higher blood lead levels in lead workers and environmentally exposed children with the 1-2 and 2-2 isozymes. Environ Res. 1991;56:109-119.
5. Battistuzzi G, Petrucci R, Silvagni L, Urbani FR, Caiola S. Delta-aminolevulinate dehydrase: a new genetic polymorphism in man. Ann Hum Genet. 1981;45:223-229.
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