A case of valproate-induced hyperammonemia due to carnitine deficiency who responded well to levocarnitine therapy-Reference-Cited by-同舟云学术

A case of valproate-induced hyperammonemia due to carnitine deficiency who responded well to levocarnitine therapy

Published:2018-08-20 Issue:8 Volume:59 Page:421-426
ISSN:0451-4203
Container-title:Kanzo
language:en
Short-container-title:Acta hepatologica Japonica

Author:

Nozu Shohei¹,Michitaka Kojiro¹²,Hiraoka Atsushi²,Aibiki Toshihiko²,Okudaira Tomonari²,Yamago Hiroka²,Iwasaki Ryuichiro²,Tomida Hideomi²,Tsubouchi Eiji²,Ninomiya Tomoyuki²

Affiliation:

1. Postgraduate Clinical Training Center, Ehime Prefectural Central Hospital

2. Department of Gastroenterology, Ehime Prefectural Central Hospital

Publisher

Japan Society of Hepatology

Subject

Hepatology

Link

https://www.jstage.jst.go.jp/article/kanzo/59/8/59_421/_pdf

Reference20 articles.

1. 1) Coulter DL, Allen RJ. Secondary hyperammonemia: A possible mechanism for valproate encephalopathy. Lancet 1980; 1: 1310-1311

2. 2) Coulter DL, Allen RJ. Hyperanmmonemia with valproic therapy. J Pediatrics 1981; 99: 317-319

3. 3) Murphy JV, Marquardt KM, Shung AL. Valproic acid associated abnormalities of carnitine metabolism. Lancet 1985; 325: 820-821

4. 4) Coulter DL. Carnitine, valproate, and toxicity. J Child Neurol 1991; 6: 7-14

5. 5) Coulter DL. Carnitine deficiency: a possible mechanism for valproate hepatotoxicity. Lancet 1984; 24: 689

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