Isoform-Specific Monoclonal Antibodies Against 3β-Hydroxysteroid Dehydrogenase/Isomerase Family Provide Markers for Subclassification of Human Primary Aldosteronism

Author:

Doi Masao1,Satoh Fumitoshi2,Maekawa Takashi3,Nakamura Yasuhiro3,Fustin Jean-Michel1,Tainaka Motomi1,Hotta Yunhong1,Takahashi Yukari1,Morimoto Ryo2,Takase Kei4,Ito Sadayoshi2,Sasano Hironobu3,Okamura Hitoshi1

Affiliation:

1. Department of Systems Biology (M.D., J.-M.F., M.T., Y.H., Y.T., H.O.), Graduate School of Pharmaceutical Sciences, Kyoto University, Sakyo-ku, Kyoto 606–8501, Japan

2. Departments of Medicine (F.S., R.M., S.I.), Sendai 980–8574, Japan

3. Pathology (T.M., Y.N., H.S.), Sendai 980–8574, Japan

4. Radiology (K.T.), Division of Nephrology, Endocrinology, and Vascular Medicine, Tohoku University Hospital, Sendai 980–8574, Japan

Publisher

The Endocrine Society

Subject

Biochemistry (medical),Clinical Biochemistry,Endocrinology,Biochemistry,Endocrinology, Diabetes and Metabolism

Reference19 articles.

1. Molecular biology of the 3β-hydroxysteroid dehydrogenase/δ5-δ4 isomerase gene family;Simard;Endocr Rev,2005

2. Salt-sensitive hypertension in circadian clock-deficient Cry-null mice involves dysregulated adrenal Hsd3b6;Doi;Nat Med,2010

3. Medicine. The genetics of primary aldosteronism;Funder;Science,2011

4. A comprehensive review of the clinical aspects of primary aldosteronism;Rossi;Nat Rev Endocrinol,2011

5. Generation of high-affinity antibody against T cell-dependent antigen in the Ganp gene-transgenic mouse;Sakaguchi;J Immunol,2005

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