Estrogen Stimulation of Kiss1 Expression in the Medial Amygdala Involves Estrogen Receptor-α But Not Estrogen Receptor-β
Author:
Affiliation:
1. Department of Reproductive Medicine, University of California, San Diego, La Jolla, California 92093
Publisher
The Endocrine Society
Subject
Endocrinology
Link
http://academic.oup.com/endo/article-pdf/157/10/4021/10841053/endo4021.pdf
Reference42 articles.
1. The GPR54 gene as a regulator of puberty;Seminara;N Engl J Med,2003
2. Inactivating KISS1 mutation and hypogonadotropic hypogonadism;Topaloglu;N Engl J Med,2012
3. Kiss1−/− mice exhibit more variable hypogonadism than Gpr54−/− mice;Lapatto;Endocrinology,2007
4. Hypogonadotropic hypogonadism due to loss of function of the KiSS1-derived peptide receptor GPR54;de Roux;Proc Natl Acad Sci USA,2003
5. Increased hypothalamic GPR54 signaling: a potential mechanism for initiation of puberty in primates;Shahab;Proc Natl Acad Sci USA,2005
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