Low-Dose Dihydrotestosterone Drives Metabolic Dysfunction via Cytosolic and Nuclear Hepatic Androgen Receptor Mechanisms
Author:
Affiliation:
1. Department of Pediatrics, Johns Hopkins School of Medicine, Baltimore, Maryland 21283
Funder
National Institutes of Health
Baltimore Diabetes Research Center
Publisher
The Endocrine Society
Subject
Endocrinology
Link
http://academic.oup.com/endo/article-pdf/158/3/531/28690986/en.2016-1553.pdf
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