Inhibin and p27 Interact to Regulate Gonadal Tumorigenesis-Reference-Cited by-同舟云学术

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Inhibin and p27 Interact to Regulate Gonadal Tumorigenesis

Published:2001-06-01 Issue:6 Volume:15 Page:985-996
ISSN:0888-8809
Container-title:Molecular Endocrinology
language:en
Short-container-title:

Author:

Cipriano Sherry C.¹,Chen Lei¹,Burns Kathleen H.¹²,Koff Andrew³,Matzuk Martin M.¹⁴²

Affiliation:

1. Departments of Pathology (S.C.C., L.C., K.H.B., M.M.M.) Texas 77030

2. Molecular and Human Genetics (K.H.B., M.M.M.) Baylor College of Medicine Houston, Texas 77030

3. Program in Molecular Biology (A.K.) Memorial Sloan-Kettering Cancer Center New York, New York 10021

4. Molecular and Cellular Biology (M.M.M.), Texas 77030

Publisher

The Endocrine Society

Subject

Endocrinology,Molecular Biology,General Medicine

Link

http://academic.oup.com/mend/article-pdf/15/6/985/10716383/mend0985.pdf

Reference46 articles.

1. Inhibitors of mammalian G1 cyclin-dependent kinases.;Sherr;Genes Dev,1995

2. Identification of human and mouse p19, a novel CDK4 and CDK6 inhibitor with homology to p16ink4.;Chan;Mol Cell Biol,1995

3. WAF1, a potential mediator of p53 tumor suppression p27, a novel inhibitor of G1 cyclin-Cdk protein kinase activity, is related to p21.;el-Deiry;Cell,1993

4. Growth suppression by p18, a p16INK4/MTS1- and p14INK4B/MTS2-related CDK6 inhibitor, correlates with wild-type pRb function.;Guan;Genes Dev,1994

5. p15INK4B is a potential effector of TGF-β-induced cell cycle arrest.;Hannon;Nature,1994

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