Lack of B7-1/BB1 and B7-2/B70 expression on thyrocytes of patients with Graves' disease. Delivery of costimulatory signals from bystander professional antigen-presenting cells
Author:
Publisher
The Endocrine Society
Subject
Biochemistry (medical),Clinical Biochemistry,Endocrinology,Biochemistry,Endocrinology, Diabetes and Metabolism
Cited by 32 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. CD28/CTLA-4/ICOS haplotypes confers susceptibility to Graves’ disease and modulates clinical phenotype of disease;Endocrine;2016-09-16
2. Influence of Iodide Excess and Interferon-γ on Human Primary Thyroid Cell Proliferation, Thyroglobulin Secretion, and Intracellular Adhesion Molecule-1 and Human Leukocyte Antigen-DR Expression;Thyroid;2009-03
3. Association of Graves’ Disease and Prevalence of Circulating IFN-γ-producing CD28− T Cells;Journal of Clinical Immunology;2008-08-14
4. Thyroid Cancers Express CD-40 and CD-40 Ligand: Cancers That Express CD-40 Ligand May Have a Greater Risk of Recurrence in Young Patients;Thyroid;2005-02
5. Relationship between CTLA-4 and CD28 Molecule Expression on T Lymphocytes and Stimulating and Blocking Autoantibodies to the TSH-Receptor in Children with Graves’ Disease;Hormone Research in Paediatrics;2005
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