The Paradoxical Increase in Cortisol Secretion Induced by Dexamethasone in Primary Pigmented Nodular Adrenocortical Disease Involves a Glucocorticoid Receptor-Mediated Effect of Dexamethasone on Protein Kinase A Catalytic Subunits-Reference-Cited by-同舟云学术

The Paradoxical Increase in Cortisol Secretion Induced by Dexamethasone in Primary Pigmented Nodular Adrenocortical Disease Involves a Glucocorticoid Receptor-Mediated Effect of Dexamethasone on Protein Kinase A Catalytic Subunits

Published:2009-07-01 Issue:7 Volume:94 Page:2406-2413
ISSN:0021-972X
Container-title:The Journal of Clinical Endocrinology & Metabolism
language:en
Short-container-title:

Author:

Louiset Estelle¹,Stratakis Constantine A.²,Perraudin Véronique¹,Griffin Kurt J.²,Libé Rossella³,Cabrol Sylvie⁴,Fève Bruno⁵,Young Jacques⁵,Groussin Lionel³,Bertherat Jérôme³,Lefebvre Hervé¹⁶

Affiliation:

1. Institut National de la Santé et de la Recherche Médicale (INSERM) Unité 413, EA4310, L’Institut Fédératif de Recherches Multidisciplinaires sur les Peptides 23, Laboratory of Differentiation and Neuronal and Neuroendocrine Communication (E.L., V.P., H.L.), University of Rouen, Mont Saint Aignan, 76821, France

2. Section on Endocrinology and Genetics, Program in Developmental Endocrinology and Genetics (C.A.S., K.J.G.), National Institute of Child Health and Human Development, Bethesda, Maryland 20892

3. Department of Endocrinology (R.L., L.G., J.B.), Centre Hospitalier Universitaire Cochin and Institut Cochin, INSERM U567, Centre National de la Recherche Scientifique Unité Mixte de Recherche 8104, Institut Fédératif de Recherche 116, Université Paris V-René Descartes, 75014 Paris, France

4. Department of Pediatric Endocrinology (S.C.), University Paris VI-Pierre-et-Marie-Curie, Hospital Trousseau, 75571 Paris, France

5. Department of Endocrinology (B.F., J.Y.), University Paris-Sud 11, Hospital Bicêtre, 94275 Le Kremlin Bicêtre, France

6. Department of Endocrinology (H.L.), University Hospital of Rouen, Institute for Biomedical Research, University of Rouen, 76031Rouen, France

Publisher

The Endocrine Society

Subject

Biochemistry (medical),Clinical Biochemistry,Endocrinology,Biochemistry,Endocrinology, Diabetes and Metabolism

Link

http://academic.oup.com/jcem/article-pdf/94/7/2406/9065049/jcem2406.pdf

Reference24 articles.

1. Clinical and genetic analysis of primary bilateral adrenal diseases (micro- and macronodular disease) leading to Cushing syndrome.;Stratakis;Horm Metab Res,1998

2. Mutations in regulatory subunit type 1A of cyclic AMP-dependent protein kinase (PRKAR1A): phenotype analysis in 353 patients and 80 different genotypes.;Bertherat;J Clin Endocrinol Metab,17 2009

3. A PRKAR1A mutation associated with primary pigmented nodular adrenocortical disease in 12 kindreds.;Groussin;J Clin Endocrinol Metab,2006

4. Molecular analysis of the cyclic AMP-dependent protein kinase A (PKA) regulatory subunit 1A (PRKAR1A) gene in patients with Carney complex and primary pigmented nodular adrenocortical disease (PPNAD) reveals novel mutations and clues for pathophysiology: augmented PKA signaling is associated with adrenal tumorigenesis in PPNAD.;Groussin;Am J Hum Genet,2002

5. Genetics of adrenal tumors associated with Cushing’s syndrome: a new classification for bilateral adrenocortical hyperplasias.;Stratakis;Nat Clin Pract Endocrinol Metab,2007

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