Fos family members successively occupy the tyrosine hydroxylase gene AP- 1 site after nerve growth factor or epidermal growth factor stimulation and can repress transcription
Author:
Publisher
The Endocrine Society
Subject
Endocrinology,Molecular Biology,General Medicine
Cited by 46 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. CD38/cADPR/Ca2+ Pathway Promotes Cell Proliferation and Delays Nerve Growth Factor-induced Differentiation in PC12 Cells;Journal of Biological Chemistry;2009-10
2. Nerve Growth Factor Regulation of Cyclin D1 in PC12 Cells through a p21RASExtracellular Signal-regulated Kinase Pathway Requires Cooperative Interactions between Sp1 and Nuclear Factor-κB;Molecular Biology of the Cell;2008-06
3. Regulation of Serine (Ser)-31 and Ser40 Tyrosine Hydroxylase Phosphorylation during Morphine Withdrawal in the Hypothalamic Paraventricular Nucleus and Nucleus Tractus Solitarius-A2 Cell Group: Role of ERK1/2;Endocrinology;2007-12-01
4. Tyrosine hydroxylase expression is unstable in a human immortalized mesencephalic cell line—Studies in vitro and after intracerebral grafting in vivo;Molecular and Cellular Neuroscience;2007-03
5. Erratum to “Cellular mechanisms underlying acquired epilepsy: The calcium hypothesis of the induction and maintenance of epilepsy” [Pharmacol. Ther. 105(3) (2005) 229–266];Pharmacology & Therapeutics;2006-07
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