Dioxin Perturbs, in a Dose- and Time-Dependent Fashion, Steroid Secretion, and Induces Apoptosis of Human Luteinized Granulosa Cells**This work was supported, in part, by Grants NIH-ES-06807 and ES-08342 (to R.J.H.) from the National Institute of Environmental Health Sciences, and the Office on Research for Women’s Health, National Institutes of Health, Research Triangle Park, North Carolina.
Author:
Affiliation:
1. Department of Biological Sciences (I.H., H.O., R.J.H.), University of Wisconsin-Milwaukee, Milwaukee, Wisconsin 53211;
2. Department of Obstetrics and Gynecology (R.G.R.), Rush Presbyterian St. Luke’s Medical Center, Chicago, Illinois 60612
Publisher
The Endocrine Society
Subject
Endocrinology
Link
http://academic.oup.com/endo/article-pdf/139/10/4373/10351904/endo4373.pdf
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3. Mechanism of action and structure-activity relationships for the chlorinated dibenzo-p-dioxins and related compounds.;Goldstein,1989
4. The aryl hydrocarbonreceptor complex.;Hankinson;Annu Rev Pharmacol Toxicol,1995
5. Identification of the Ah receptor in selected mammalian species and induction of aryl hydrocarbon hydroxylase.;Denison;Eur J Biochem,1985
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