Insulin Receptor Substrate-1 and Phosphatidylinositol 3-Kinase Regulate Extracellular Signal-Regulated Kinase-Dependent and -Independent Signaling Pathways during Myogenic Differentiation-Reference-Cited by-同舟云学术

Insulin Receptor Substrate-1 and Phosphatidylinositol 3-Kinase Regulate Extracellular Signal-Regulated Kinase-Dependent and -Independent Signaling Pathways during Myogenic Differentiation

Published:1998-12-01 Issue:12 Volume:12 Page:1870-1878
ISSN:0888-8809
Container-title:Molecular Endocrinology
language:en
Short-container-title:

Author:

Sarbassov Dos D.¹,Peterson Charlotte A.¹

Affiliation:

1. Donald W. Reynolds Department of Geriatrics and the Department of Biochemistry and Molecular Biology University of Arkansas for Medical Sciences and The Geriatric Research, Education, and Clinical Center McClellan Veterans Hospital Little Rock, Arkansas 72205

Publisher

The Endocrine Society

Subject

Endocrinology,Molecular Biology,General Medicine

Link

http://academic.oup.com/mend/article-pdf/12/12/1870/8953941/mend1870.pdf

Reference55 articles.

1. “Spontaneous” differentiation of skeletal myoblasts is dependent upon autocrine secretion of insulin-like growth factor-II.;Florini;J Biol Chem,1991

2. Insulin-like growth factor-II is an autocrine survival factor for differentiating myoblasts.;Stewart;J Biol Chem,1996

3. Overexpression of insulin-like growth factor-II induces accelerated myoblast differentiation.;Stewart;J Cell Physiol,1996

4. Specific, temporally regulated expression of the insulin-like growth factor II gene during muscle cell differentiation.;Rosen;Endocrinology,1993

5. Extracellular signal-regulated kinase-1 and -2 respond differently to mitogenic and differentiative signaling pathways in myoblasts.;Sarbassov;Mol Endocrinol,1997

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