Determination of the Elimination Half-Life of Fibroblast Growth Factor-23

Author:

Khosravi Azarmindokht1,Cutler Carolee M.1,Kelly Marilyn H.1,Chang Richard2,Royal Richard E.3,Sherry Richard M.3,Wodajo Felasfa M.4,Fedarko Neal S.5,Collins Michael T.1

Affiliation:

1. Skeletal Clinical Studies Unit (A.K., C.M.C., M.H.K., M.T.C.), Craniofacial and Skeletal Diseases Branch, National Institute of Dental and Craniofacial Research, Bethesda, Maryland 20892

2. Department of Radiology (R.C.), Mark O. Hatfield Clinical Center, Bethesda, Maryland 20892

3. Surgery Branch (R.E.R., R.M.S.), National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, Maryland 20892

4. Department of Orthopedics and Pediatrics (F.M.W.), Georgetown University, Washington, D.C.

5. Division of Geriatric Medicine and Gerontology (N.S.F.), Department of Medicine, Johns Hopkins University, Baltimore, Maryland 21224

Publisher

The Endocrine Society

Subject

Biochemistry (medical),Clinical Biochemistry,Endocrinology,Biochemistry,Endocrinology, Diabetes and Metabolism

Reference29 articles.

1. Brief report: inhibition of renal phosphate transport by a tumor product in a patient with oncogenic osteomalacia.;Cai;N Engl J Med,1994

2. Tumor-induced osteomalacia: clinical and basic studies.;Shane;J Bone Miner Res,1997

3. Tumor-induced osteomalacia.;Jan de Beur;JAMA,2005

4. Tumor-induced osteomalacia and the regulation of phosphate homeostasis.;Kumar;Bone,2000

5. Tumor-induced osteomalacia.;Drezner;Rev Endocr Metab Disord,2001

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