Pharmacokinetic Factors Contribute to the Inverse Relationship between Luteinizing Hormone and Body Mass Index in Polycystic Ovarian Syndrome

Author:

Srouji Serene S.12,Pagán Yanira L.1,D’Amato Fernando3,Dabela Amsalu1,Jimenez Yarisie1,Supko Jeffrey G.3,Hall Janet E.1

Affiliation:

1. Reproductive Endocrine Unit (S.S.S., Y.L.P., A.D., Y.J., J.E.H.), Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts 02114

2. Center for Reproductive Medicine, Department of Obstetrics and Gynecology (S.S.S.), Brigham and Women’s Hospital, Boston, Massachusetts 02115

3. Division of Hematology/Oncology (F.D., J.G.S.), Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts 02114

Publisher

The Endocrine Society

Subject

Biochemistry, medical,Clinical Biochemistry,Endocrinology,Biochemistry,Endocrinology, Diabetes and Metabolism

Reference43 articles.

1. Inappropriate secretion of follicle-stimulating hormone and luteinizing hormone in polycystic ovarian disease.;Yen;J Clin Endocrinol Metab,1970

2. Characterization of the inappropriate gonadotropin secretion in polycystic ovary syndrome.;Rebar;J Clin Invest,1976

3. Hyperfunction of the hypothalamic-pituitary axis in women with polycystic ovarian disease: indirect evidence for partial gonadotroph desensitization.;Waldstreicher;J Clin Endocrinol Metab,1988

4. Determinants of abnormal gonadotropin secretion in clinically defined women with polycystic ovary syndrome.;Taylor;J Clin Endocrinol Metab,1997

5. Inappropriate gonadotropin secretion in polycystic ovary syndrome: influence of adiposity.;Arroyo;J Clin Endocrinol Metab,1997

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