1,25-Dihydroxyvitamin D3 and 9-cis-Retinoic Acid Act Synergistically to Inhibit the Growth of LNCaP Prostate Cells and Cause Accumulation of Cells in G1*

Author:

Blutt Sarah E.1,Allegretto Elizabeth A.2,Pike J. Wesley3,Weigel Nancy L.

Affiliation:

1. Department of Cell Biology, Baylor College of Medicine (S.E.B., N.L W.), Houston, Texas 77030

2. Department of Ligand Pharmaceuticals (E.A.A.) San Diego, California 92121

3. The Department of Molecular and Cellular Physiology, University of Cincinnati (J.W.P.), Cincinnati, Ohio 45267

Publisher

The Endocrine Society

Subject

Endocrinology

Reference31 articles.

1. Is vitamin D deficiency a risk factor for prostate cancer? (Hypothesis).;Schwartz;Anticancer Res,1990

2. Antiproliferative effects of 1,25-dihydroxyvitamin D3 on primary cultures of human prostatic cells.;Peehl;Cancer Res,1994

3. Vitamin D and prostate cancer: 1,25-dihydroxyvitamin D3 receptors and actions in human prostate cancer cell lines.;Skowronski;Endocrinology,1993

4. Retinoic acid receptors and cellular retinoid binding proteins: complex interplay in retinoid signaling.;Giguere;Endocr Rev,1994

5. Molecular cloning of complementary DNA encoding the avian receptor for vitamin D.;McDonnell;Science,1987

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