Deletion of the RIIβ-Subunit of Protein Kinase A Decreases Body Weight and Increases Energy Expenditure in the Obese, Leptin-Deficient ob/ob Mouse-Reference-Cited by-同舟云学术

Deletion of the RIIβ-Subunit of Protein Kinase A Decreases Body Weight and Increases Energy Expenditure in the Obese, Leptin-Deficient ob/ob Mouse

Published:2005-04-01 Issue:4 Volume:19 Page:982-991
ISSN:0888-8809
Container-title:Molecular Endocrinology
language:en
Short-container-title:

Author:

Newhall Kathryn J.¹,Cummings David E.²,Nolan Michael A.³,McKnight G. Stanley¹

Affiliation:

1. Department of Pharmacology (K.J.N, G.S.M.), Seattle, Washington 98195;

2. University of Washington, and Division of Metabolism (D.E.C.), Endocrinology and Nutrition, University of Washington, Veterans Affairs Puget Sound Health Care System, Seattle, Washington 98195;

3. Wyeth Research (M.A.N.), Collegeville, Pennsylvania 19426

Publisher

The Endocrine Society

Subject

Endocrinology,Molecular Biology,General Medicine

Link

http://academic.oup.com/mend/article-pdf/19/4/982/8951990/mend0982.pdf

Reference41 articles.

1. The role of uncoupling protein 1 in the metabolism and adiposity of RIIβ-protein kinase A-deficient mice.;Nolan;Mol Endocrinol,2004

2. Genetically lean mice result from targeted disruption of the RII β subunit of protein kinase A.;Cummings;Nature,1996

3. Compensatory regulation of RIα protein levels in protein kinase A mutant mice.;Amieux;J Biol Chem,1997

4. Mutation of the RIIβ subunit of protein kinase A differentially affects lipolysis but not gene induction in white adipose tissue.;Planas;J Biol Chem,1999

5. Adaptive thermogenesis: turning on the heat.;Lowell;Curr Biol,1998

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