PNPLA3 Inhibition: Replacing One Evil With the Other?
Author:
Affiliation:
1. Institute of Clinical Chemistry, University of Zurich and University Hospital Zurich , CH 8091 Zurich , Switzerland
Publisher
The Endocrine Society
Link
https://academic.oup.com/jcem/advance-article-pdf/doi/10.1210/clinem/dgae377/58181618/dgae377.pdf
Reference8 articles.
1. Metabolic dysfunction-associated steatotic liver disease: an opportunity for collaboration between cardiology and hepatology;Raggi;Atherosclerosis,2024
2. Prioritising genetic findings for drug target identification and validation;Hukerikar;Atherosclerosis,2024
3. LDL-C and TC mediate the risk of PNPLA3 inhibition on cardiovascular diseases;Zhang;J Clin Endocrinol Metab
4. The fatty liver disease-causing protein PNPLA3-I148M alters lipid droplet-Golgi dynamics;Sherman;Proc Natl Acad Sci U S A,2024
5. The PNPLA3-I148M variant confers an antiatherogenic lipid profile in insulin-resistant patients;Luukkonen;J Clin Endocrinol Metab,2021
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