Sexually Dimorphic Increases in Bone Mass Following Tissue-specific Overexpression of Runx1 in Osteoclast Precursors
Author:
Affiliation:
1. Department of Orthopaedics, Emory University School of Medicine , Atlanta, Georgia 30329 , USA
2. Atlanta VA Health Care System , Decatur, Georgia, 30033 , USA
3. Department of Medicine, UConn Health , Farmington, 06032, Connecticut , USA
Abstract
Funder
National Institutes of Health
Publisher
The Endocrine Society
Subject
Endocrinology
Link
https://academic.oup.com/endo/advance-article-pdf/doi/10.1210/endocr/bqac113/45067994/bqac113.pdf
Reference32 articles.
1. Bone health and osteoporosis: a report of the surgeon general;Office of the Surgeon General.,2004
2. Runx1 up-regulates chondrocyte to osteoblast lineage commitment and promotes bone formation by enhancing both chondrogenesis and osteogenesis;Tang;Biochem J.,2020
3. Runx1 dose-dependently regulates endochondral ossification during skeletal development and fracture healing;Soung;J Bone Miner Res.,2012
4. Runx1/AML1/Cbfa2 mediates onset of mesenchymal cell differentiation toward chondrogenesis;Wang;J Bone Miner Res.,2005
5. Runx1 is a central regulator of osteogenesis for bone homeostasis by orchestrating BMP and WNT signaling pathways;Tang;PLoS Genet.,2021
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