Pioglitazone and Sodium Salicylate Protect Human β-Cells against Apoptosis and Impaired Function Induced by Glucose and Interleukin-1β-Reference-Cited by-同舟云学术

Pioglitazone and Sodium Salicylate Protect Human β-Cells against Apoptosis and Impaired Function Induced by Glucose and Interleukin-1β

Published:2004-10-01 Issue:10 Volume:89 Page:5059-5066
ISSN:0021-972X
Container-title:The Journal of Clinical Endocrinology & Metabolism
language:en
Short-container-title:

Author:

Zeender E.¹²,Maedler K.³,Bosco D.²,Berney T.²,Donath M. Y.³,Halban P. A.¹

Affiliation:

1. Department of Genetic Medicine and Development (E.Z., P.A.H.), Geneva CH-1211, Switzerland

2. University Medical Centre and Division of Surgical Research (E.Z., T.B., D.B.), Department of Surgery, University Hospital, Geneva CH-1211, Switzerland

3. Division of Endocrinology and Diabetes (K.M., M.Y.D.), University Hospital, Zurich CH-8091, Switzerland

Publisher

The Endocrine Society

Subject

Biochemistry, medical,Clinical Biochemistry,Endocrinology,Biochemistry,Endocrinology, Diabetes and Metabolism

Link

http://academic.oup.com/jcem/article-pdf/89/10/5059/10771829/jcem5059.pdf

Reference76 articles.

1. The relative contributions of insulin resistance and β-cell dysfunction to the pathophysiology of type 2 diabetes.;Kahn;Diabetologia,2003

2. Decreased β-cell mass in diabetes: significance, mechanisms and therapeutic implications.;Donath;Diabetologia,2004

3. β-Cell deficit and increased β-cell apoptosis in humans with type 2 diabetes.;Butler;Diabetes,2003

4. Glucose-induced β-cell production of IL-1β contributes to glucotoxicity in human pancreatic islets.;Maedler;J Clin Invest,2002

5. Glucose induces β-cell apoptosis via up-regulation of the Fas receptor in human islets.;Maedler;Diabetes,2001

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