Rationale and Evidence for Sunitinib in the Treatment of Malignant Paraganglioma/Pheochromocytoma-Reference-Cited by-同舟云学术

Rationale and Evidence for Sunitinib in the Treatment of Malignant Paraganglioma/Pheochromocytoma

Published:2009-01-01 Issue:1 Volume:94 Page:5-9
ISSN:0021-972X
Container-title:The Journal of Clinical Endocrinology & Metabolism
language:en
Short-container-title:

Author:

Joshua Anthony M.¹,Ezzat Shereen¹,Asa Sylvia L.²,Evans Andrew²,Broom Reuben¹,Freeman Marc³,Knox Jennifer J.¹

Affiliation:

1. Department of Medical Oncology (A.M.J., S.E., R.B., J.J.K.), Princess Margaret Hospital, University Health Network, M5G 2M9 Toronto, Ontario, Canada

2. Departments of Pathology (S.L.A., A.E.), University Health Network, M5G 2C4, Toronto, Ontario, Canada

3. Radiology (M.F.), University Health Network, M5G 2C4, Toronto, Ontario, Canada

Publisher

The Endocrine Society

Subject

Biochemistry, medical,Clinical Biochemistry,Endocrinology,Biochemistry,Endocrinology, Diabetes and Metabolism

Link

http://academic.oup.com/jcem/article-pdf/94/1/5/9056370/jcem0005.pdf

Reference27 articles.

1. Prevalence of SDHB, SDHC, and SDHD germline mutations in clinic patients with head and neck paragangliomas.;Baysal;J Med Genet,2002

2. Mutations in SDHD, a mitochondrial complex II gene, in hereditary paraganglioma.;Baysal;Science,2004

3. Germline SDHD mutation in familial phaeochromocytoma.;Astuti;Lancet,2001

4. Gene mutations in the succinate dehydrogenase subunit SDHB cause susceptibility to familial pheochromocytoma and to familial paraganglioma.;Astuti;Am J Hum Genet [Erratum (2002) 70:565],2001

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