Testicular Effects of Isolated Luteinizing Hormone Deficiency and Reversal by Long-Term Human Chorionic Gonadotropin Treatment

Author:

Valdes-Socin Hernan1,Salvi Roberto2,Thiry Albert3,Daly Adrian F.1,Pralong François P.2,Gaillard Rolf2,Beckers Albert1

Affiliation:

1. Departments of Endocrinology (H.V.-S., A.F.D., A.B.), Centre Hospitalier Universitaire de Liège, University of Liège, 4000 Liège, Belgium

2. Division of Endocrinology, Diabetology, and Metabolism (R.S., F.P.P., R.G.), University Hospital, 1011 Lausanne, Switzerland

3. Pathology (A.T.), Centre Hospitalier Universitaire de Liège, University of Liège, 4000 Liège, Belgium

Publisher

The Endocrine Society

Subject

Biochemistry (medical),Clinical Biochemistry,Endocrinology,Biochemistry,Endocrinology, Diabetes and Metabolism

Reference5 articles.

1. Hypogonadism caused by a single amino acid substitution in the β-subunit of luteinizing hormone.;Weiss;N Engl J Med,1992

2. Hypogonadism in a male with immunologically active, biologically inactive luteinizing hormone: an exception to a venerable rule.;Axelrod;J Clin Endocrinol Metab,1979

3. Hypogonadism in a patient with a mutation in the luteinizing β-subunit gene.;Valdes-Socin;N Engl J Med,2004

4. Identification of a family harbouring a novel LH subunit mutation associated with hypogonadism. OR52-5;Daly

5. Luteinizing hormone β mutation and hypogonadism in men and woman;Lofrano-Porto;N Engl J Med.,2007

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