Increased Production of 17β-Estradiol in Endometriosis Lesions Is the Result of Impaired Metabolism-Reference-Cited by-同舟云学术

Increased Production of 17β-Estradiol in Endometriosis Lesions Is the Result of Impaired Metabolism

Published:2009-03-01 Issue:3 Volume:94 Page:876-883
ISSN:0021-972X
Container-title:The Journal of Clinical Endocrinology & Metabolism
language:en
Short-container-title:

Author:

Delvoux Bert¹²,Groothuis Patrick¹,D'Hooghe Thomas³,Kyama Cleophas³,Dunselman Gerard²,Romano Andrea¹²

Affiliation:

1. GROW (B.D., P.G., A.R.), University Hospital Maastricht, 6202 AZ Maastricht, The Netherlands

2. School for Oncology and Developmental Biology, University Maastricht, and Department of Obstetrics and Gynecology (B.D., G.D., A.R.), University Hospital Maastricht, 6202 AZ Maastricht, The Netherlands

3. Department of Obstetrics and Gynecology (T.D., C.K.), University Hospital Gasthuisberg, 3000 Leuven, Belgium

Publisher

The Endocrine Society

Subject

Biochemistry (medical),Clinical Biochemistry,Endocrinology,Biochemistry,Endocrinology, Diabetes and Metabolism

Link

http://academic.oup.com/jcem/article-pdf/94/3/876/9063530/jcem0876.pdf

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3. Endometriosis: role of ovarian steroids in initiation, maintenance, and suppression.;Dizerega;Fertil Steril,1980

4. Effect of estradiol and progesterone on human endometrial aromatase activity in primary cell culture.;Tseng;Endocrinology,1984

5. Aromatase expression in endometriosis.;Noble;J Clin Endocrinol Metab,1996

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