Human islets chronically exposed in vitro to different stimuli become unresponsive to the same stimuli given acutely: evidence supporting specific desensitization rather than beta-cell exhaustion
Author:
Publisher
The Endocrine Society
Subject
Biochemistry, medical,Clinical Biochemistry,Endocrinology,Biochemistry,Endocrinology, Diabetes and Metabolism
Link
http://academic.oup.com/jcem/article-pdf/74/4/790/10501464/jcem0790.pdf
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1. Angiotensin II exerts glucose-dependent effects on Kvcurrents in mouse pancreatic β-cells via angiotensin II type 2 receptors;American Journal of Physiology-Cell Physiology;2010-02
2. Ketosis-Prone Type 2 Diabetes: Effect of Hyperglycemia on β-Cell Function and Skeletal Muscle Insulin Signaling;Endocrine Practice;2007-05
3. The E23K Variant of KCNJ11 Encoding the Pancreatic β-Cell Adenosine 5′-Triphosphate-Sensitive Potassium Channel Subunit Kir6.2 Is Associated with an Increased Risk of Secondary Failure to Sulfonylurea in Patients with Type 2 Diabetes;The Journal of Clinical Endocrinology & Metabolism;2006-06-01
4. Increased glucose sensitivity of both triggering and amplifying pathways of insulin secretion in rat islets cultured for 1 wk in high glucose;American Journal of Physiology-Endocrinology and Metabolism;2004-08
5. Clinical islet cell transplantation;International Journal of Pancreatology;1998-12
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