Prenatal Exposure to Bisphenol A Disrupts Naturally Occurring Bimodal DNA Methylation at Proximal Promoter of fggy, an Obesity-Relevant Gene Encoding a Carbohydrate Kinase, in Gonadal White Adipose Tissues of CD-1 Mice-Reference-Cited by-同舟云学术

Prenatal Exposure to Bisphenol A Disrupts Naturally Occurring Bimodal DNA Methylation at Proximal Promoter of fggy, an Obesity-Relevant Gene Encoding a Carbohydrate Kinase, in Gonadal White Adipose Tissues of CD-1 Mice

Published:2017-12-06 Issue:2 Volume:159 Page:779-794
ISSN:1945-7170
Container-title:Endocrinology
language:en
Short-container-title:

Author:

Taylor Julia A¹,Shioda Keiko²,Mitsunaga Shino²,Yawata Shiomi²,Angle Brittany M¹,Nagel Susan C³,vom Saal Frederick S¹,Shioda Toshi²⁴

Affiliation:

1. Division of Biological Sciences, University of Missouri, Columbia, Missouri

2. Massachusetts General Hospital Center for Cancer Research, Charlestown, Massachusetts

3. Department of Obstetrics, Gynecology and Women’s Health, University of Missouri, Columbia, Missouri

4. Department of Medicine, Harvard Medical School, Boston, Massachusetts

Publisher

The Endocrine Society

Subject

Endocrinology

Link

http://academic.oup.com/endo/article-pdf/159/2/779/23372962/en.2017-00711.pdf

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4. Endocrine disrupting chemicals in mixture and obesity, diabetes and related metabolic disorders;Le Magueresse-Battistoni;World J Biol Chem,2017

5. Bisphenol-A exposure in utero programs a sexually dimorphic estrogenic state of hepatic metabolic gene expression;Ilagan;Reprod Toxicol,2017

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