Survival of steroid 21-hydroxylase-deficient mice without endogenous corticosteroids after neonatal treatment and genetic rescue by transgenesis as a model system for treatment of congenital adrenal hyperplasia in humans.
Author:
Publisher
The Endocrine Society
Subject
Endocrinology
Cited by 14 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. A Humanized and Viable Animal Model for Congenital Adrenal Hyperplasia–CYP21A2-R484Q Mutant Mouse;International Journal of Molecular Sciences;2024-05-07
2. Genetic Disruption of cyp21a2 Leads to Systemic Glucocorticoid Deficiency and Tissues Hyperplasia in the Teleost Fish Medaka (Oryzias latipes);Zoological Science;2024-04-08
3. Animal models of adrenal genetic disorders;Genetic Steroid Disorders;2023
4. cyp21a2 Knockout Tadpoles Survive Metamorphosis Despite Low Corticosterone;Endocrinology;2022-10-27
5. Towards novel treatments for adrenal diseases: Cell- and gene therapy-based approaches;Molecular and Cellular Endocrinology;2021-03
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