Persistent Activation of Phosphatidylinositol 3-Kinase Causes Insulin Resistance Due to Accelerated Insulin-Induced Insulin Receptor Substrate-1 Degradation in 3T3-L1 Adipocytes*-Reference-Cited by-同舟云学术

Persistent Activation of Phosphatidylinositol 3-Kinase Causes Insulin Resistance Due to Accelerated Insulin-Induced Insulin Receptor Substrate-1 Degradation in 3T3-L1 Adipocytes*

Published:2000-06-01 Issue:6 Volume:141 Page:1930-1935
ISSN:0013-7227
Container-title:Endocrinology
language:en
Short-container-title:

Author:

Egawa Katsuya¹²,Nakashima Naoki²,Sharma Prem M.²,Maegawa Hiroshi¹,Nagai Yoshio¹,Kashiwagi Atsunori¹,Kikkawa Ryuichi¹,Olefsky Jerrold M.³⁴²

Affiliation:

1. Third Department of Medicine (K.E., H.M., Y.N., A.K., R.K.), Shiga University of Medical Science, Otsu, Shiga 520-2192, Japan

2. Department of Medicine (K.E., N.N., P.M.S., J.M.O.), Division of Endocrinology and Metabolism, University of California, San Diego, La Jolla, California 92093

3. Department of The Whittier Diabetes Institute (J.M.O.), University of California, San Diego, La Jolla, California 92093

4. Department of Veterans Administration Research Service (J.M.O.), San Diego, La Jolla, California 92161

Publisher

The Endocrine Society

Subject

Endocrinology

Link

http://academic.oup.com/endo/article-pdf/141/6/1930/10359521/endo1930.pdf

Reference21 articles.

1. A novel 160-kDa phosphotyrosine protein in insulin-treated embryonic kidney cells is a new member of the insulin receptor substrate family.;Lavan;J Biol Chem,1997

2. The 60-kDa phosphotyrosine protein in insulin-treated adipocytes is a new member of the insulin receptor substrate family.;Lavan;J Biol Chem,1997

3. The insulin signaling system.;White;J Biol Chem,1994