Annexin 1 (Lipocortin 1) Mediates the Glucocorticoid Inhibition of Cyclic Adenosine 3′,5′-Monophosphate-Stimulated Prolactin Secretion*

Author:

Taylor A. D.1,Philip J. G.1,John C. D.1,Cover P. O.1,Morris J. F.1,Flower R. J.2,Buckingham J. C.1

Affiliation:

1. Department of Neuroendocrinology, Division of Neuroscience and Psychological Medicine, Imperial College School of Medicine, Charing Cross Hospital, London, United Kingdom W6 8RF; Department of Human Anatomy and Genetics, University of Oxford (J.F.M.), Oxford, United Kingdom OX1 3QX;

2. Department of Biochemical Pharmacology, The William Harvey Research Institute, St. Bartholomew’s and The Royal London School of Medicine at Queen Mary and Westfield College (R.J.F.), London, United Kingdom EC1M 6BQ

Publisher

The Endocrine Society

Subject

Endocrinology

Reference48 articles.

1. Central nervous system-immune interactions: psychoneuroendocrinology of stress and its immune consequences.;Black;Antimicrobial Agents Chemother,1994

2. Role of classic and peptide neuromediators in the neuroendocrine regulation of LH and prolactin;Weiner;In: Knobil E, Neill JD (ed) The Physiology of Reproduction. Raven Press, New York, pp,1988

3. Effect of corticosterone on the prolactin response to psychological and physical stress in rats.;Yelvington;Life Sci,1984

4. The neuroendocrine system: anatomy, physiology and responses to stress;Buckingham;In: Buckingham JC, Cowell A-M, Gillies GE (ed) Stress, Stress Hormones and the Immune System. Wiley & Sons, pp,1997

5. Effects of glucocorticoids on pituitary hormonal responses to hypoglycaemia. Inhibition of prolactin release.;Copinschi;J Clin Endocrinol Metab,1975

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