Osteopontin Facilitates Angiogenesis, Accumulation of Osteoclasts, and Resorption in Ectopic Bone*

Author:

Asou Yoshinori1,Rittling Susan R.2,Yoshitake Hiroyuki1,Tsuji Kunikazu1,Shinomiya Kenichi3,Nifuji Akira1,Denhardt David T.2,Noda Masaki1

Affiliation:

1. Department of Molecular Pharmacology, Medical Research Institute (Y.A., H.Y., K.T., A.N., M.N.), Tokyo Medical and Dental University, Tokyo 101-0062, Japan

2. Department of Cell Biology and Neuroscience, Rutgers University (S.R.R., D.T.D.), Piscataway, New Jersey 08854

3. Department of Orthopedic Surgery (K.S.), Tokyo Medical and Dental University, Tokyo 101-0062, Japan

Publisher

The Endocrine Society

Subject

Endocrinology

Reference33 articles.

1. Osteopontin-deficient mice are resistant to ovariectomy-induced bone resorption [published erratum appears in Proc Natl Acad Sci USA 1999;Yoshitake;96:10944]. Proc Natl Acad Sci. USA.,1999

2. Expression and distribution of osteopontin in human tissues: widespread association with luminal epithelial surfaces.;Brown;Mol Biol Cell,1992

3. Osteopontin expression and function: role in bone remodeling.;Denhardt;J Cell Biochem [Suppl],1998

4. Osteopontin–a possible anchor of osteoclasts to bone.;Reinholt;Proc Natl Acad Sci USA,1990

5. Inhibition of Arg-Gly-Asp (RGD)-mediated cell adhesion to osteopontin by a monoclonal antibody against osteopontin.;Bautista;J Biol Chem,1994

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