β-Cell-Targeted Expression of a Dominant-Negative Hepatocyte Nuclear Factor-1α Induces a Maturity-Onset Diabetes of the Young (MODY)3-Like Phenotype in Transgenic Mice-Reference-Cited by-同舟云学术

β-Cell-Targeted Expression of a Dominant-Negative Hepatocyte Nuclear Factor-1α Induces a Maturity-Onset Diabetes of the Young (MODY)3-Like Phenotype in Transgenic Mice

Published:2001-12-01 Issue:12 Volume:142 Page:5311-5320
ISSN:0013-7227
Container-title:Endocrinology
language:en
Short-container-title:

Author:

Hagenfeldt-Johansson Kerstin A.¹,Herrera Pedro L.²,Wang Haiyan¹,Gjinovci Asllan¹,Ishihara Hisamitsu¹,Wollheim Claes B.¹

Affiliation:

1. Division of Clinical Biochemistry, Department of Internal Medicine (K.A.H.-J., H.W., A.G., H.I., C.B.W.), University Medical Center, 1211 Geneva 4, Switzerland

2. Department of Morphology (P.L.H.), University Medical Center, 1211 Geneva 4, Switzerland

Publisher

The Endocrine Society

Subject

Endocrinology

Link

http://academic.oup.com/endo/article-pdf/142/12/5311/10364845/endo5311.pdf

Reference32 articles.

1. Maturity-onset diabetes of the young: clinical heterogeneity explained by genetic heterogeneity.;Hattersley;Diabet Med,1998

2. Characterization of the MODY3 phenotype. Early-onset diabetes caused by an insulin secretion defect.;Lehto;J Clin Invest,1997

3. Glucose utilization and production in patients with maturity-onset diabetes of the young caused by a mutation of the hepatocyte nuclear factor-1α gene.;Surmely;Diabetes,1998

4. Altered insulin secretory responses to glucose in diabetic and nondiabetic subjects with mutations in the diabetes susceptibility gene MODY3 on chromosome 12.;Byrne;Diabetes,1996