Low TSH Requirement and Goiter in Transgenic Mice Overexpressing IGF-I and IGF-I Receptor in the Thyroid Gland-Reference-Cited by-同舟云学术

Low TSH Requirement and Goiter in Transgenic Mice Overexpressing IGF-I and IGF-I Receptor in the Thyroid Gland

Published:2001-12-01 Issue:12 Volume:142 Page:5131-5139
ISSN:0013-7227
Container-title:Endocrinology
language:en
Short-container-title:

Author:

Clément Serge¹,Refetoff Samuel²,Robaye Bernard¹,Dumont Jacques E.¹,Schurmans Stéphane¹

Affiliation:

1. Institut de Recherche Interdisciplinaire en Biologie Humaine et Nucléaire, Institut de Biologie et de Médecine Moléculaire, Université Libre de Bruxelles (S.C., B.R., J.E.D., S.S.), 6041 Gosselies, Belgium

2. Departments of Medicine and Pediatrics, University of Chicago (S.R.), Chicago, Illinois 60637

Publisher

The Endocrine Society

Subject

Endocrinology

Link

http://academic.oup.com/endo/article-pdf/142/12/5131/10362472/endo5131.pdf

Reference28 articles.

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2. The thyrotropin receptor and the regulation of thyrocyte function and growth.;Vassart;Endocr Rev,1992

3. Thyrotropin via cyclic AMP induces insulin receptor expression and insulin co-stimulation of growth and amplifies insulin and insulin-like growth factor signaling pathways in dog thyroid epithelial cells.;Burikhanov;J Biol Chem,1996

4. IGF-I or insulin, and the TSH cyclic AMP cascade separately control dog and human thyroid cell growth and DNA synthesis, and complement each other in inducing mitogenesis.;Deleu;Mol Cell Endocrinol,1999

5. Thyroglobulin gene expression is regulated by insulin and insulin-like growth factor I, as well as thyrotropin, in FRTL-5 thyroid cells.;Santisteban;J Biol Chem,1987

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