Low TSH Requirement and Goiter in Transgenic Mice Overexpressing IGF-I and IGF-I Receptor in the Thyroid Gland

Author:

Clément Serge1,Refetoff Samuel2,Robaye Bernard1,Dumont Jacques E.1,Schurmans Stéphane1

Affiliation:

1. Institut de Recherche Interdisciplinaire en Biologie Humaine et Nucléaire, Institut de Biologie et de Médecine Moléculaire, Université Libre de Bruxelles (S.C., B.R., J.E.D., S.S.), 6041 Gosselies, Belgium

2. Departments of Medicine and Pediatrics, University of Chicago (S.R.), Chicago, Illinois 60637

Publisher

The Endocrine Society

Subject

Endocrinology

Reference28 articles.

1. Physiological and pathological regulation of thyroid cell proliferation and differentiation by thyrotropin and others factors.;Dumont;Physiol Rev,1992

2. The thyrotropin receptor and the regulation of thyrocyte function and growth.;Vassart;Endocr Rev,1992

3. Thyrotropin via cyclic AMP induces insulin receptor expression and insulin co-stimulation of growth and amplifies insulin and insulin-like growth factor signaling pathways in dog thyroid epithelial cells.;Burikhanov;J Biol Chem,1996

4. IGF-I or insulin, and the TSH cyclic AMP cascade separately control dog and human thyroid cell growth and DNA synthesis, and complement each other in inducing mitogenesis.;Deleu;Mol Cell Endocrinol,1999

5. Thyroglobulin gene expression is regulated by insulin and insulin-like growth factor I, as well as thyrotropin, in FRTL-5 thyroid cells.;Santisteban;J Biol Chem,1987

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