Defective Bone Formation and Anabolic Response to Exogenous Estrogen in Mice with Targeted Disruption of Endothelial Nitric Oxide Synthase**This study was supported by grants from the Arthritis Research Campaign (UK) and the Medical Research Council (UK).
Author:
Affiliation:
1. Department of Medicine and Therapeutics, Foresterhill, University of Aberdeen Medical School, Aberdeen, AB25 2ZD, United Kingdom; and Institüt für Herz- und Kreislaufphysiologie (A.G.), Heinrich-Heine-Universität, 40225 Düsseldorf, Germany
Publisher
The Endocrine Society
Subject
Endocrinology
Link
http://academic.oup.com/endo/article-pdf/142/2/760/10364843/endo0760.pdf
Reference41 articles.
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3. Inducible production of nitric oxide in osteoblast like cells and in fetal bone explants is associated with supression of osteoclastic bone resorption.;Lowik;J Clin Invest,1994
4. Mechanisms of cytokine induced bone resorption: role of nitric oxide, cyclic guanosine monophosphate and prostaglandins.;Ralston;Bone,1996
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