S179D-Human PRL, a Pseudophosphorylated Human PRL Analog, Is an Agonist and Not an Antagonist-Reference-Cited by-同舟云学术

S179D-Human PRL, a Pseudophosphorylated Human PRL Analog, Is an Agonist and Not an Antagonist

Published:2001-09-01 Issue:9 Volume:142 Page:3950-3963
ISSN:0013-7227
Container-title:Endocrinology
language:en
Short-container-title:

Author:

Bernichtein Sophie¹,Kinet Sandrina¹²,Jeay Sébastien¹,Llovera Marta¹,Madern Dominique³,Martial Joseph A.²,Kelly Paul A.¹,Goffin Vincent¹

Affiliation:

1. INSERM, U-344, Molecular Endocrinology, Faculté de Médecine Necker (S.B., S.K., S.J., M.L., P.A.K., V.G.), 75730 Paris, France

2. Laboratory of Molecular Biology and Genetic Engineering, University of Liege (S.K., J.A.M.), 4000 Sart-Tilman, Belgium

3. Laboratory of Molecular Biophysics, Institut de Biologie Structurale, Commissariat à l’énergie atomique-Centre National de la Recherche Scientifique (D.M.), 38027 Grenoble, France

Publisher

The Endocrine Society

Subject

Endocrinology

Link

http://academic.oup.com/endo/article-pdf/142/9/3950/10367451/endo3950.pdf

Reference59 articles.

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2. Defective mammopoiesis, but normal hematopoiesis, in mice with a targeted disruption of the prolactin gene.;Horseman;EMBO J,1997

3. Null mutation of the prolactin receptor gene produces multiple reproductive defects in the mouse.;Ormandy;Genes Dev,1997

4. The prolactin/growth hormone receptor family.;Kelly;Endocr Rev,1991

5. Prolactin and growth hormone receptors.;Goffin;Clin Endocrinol (Oxf),1996

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